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Adipose-specific SAT1 knockout mice develop late-onset obesity.

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Last updated 24 fevereiro 2025
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Specific knockout Akt1 in BAT increases Ucp1 expression. (a) Body
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Adipose-specific SAT1 knockout mice develop late-onset obesity
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Nicotinamide N-methyltransferase knockdown protects against diet
Adipose-specific SAT1 knockout mice develop late-onset obesity.
BCAA catabolism drives adipogenesis via an intermediate metabolite
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Multiomics reveals persistence of obesity-associated immune cell
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Identification of functionally distinct fibro-inflammatory and
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Key protein linked to appetite and obesity in mice
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Growth of A/J and C57BL/6J mice weaned onto low-or high-fat diets
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Susceptibility to diet-induced obesity at thermoneutral conditions
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Fang Yuan's research works University of California, Irvine, CA
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Brown Adipose Expansion and Remission of Glycemic Dysfunction in
Adipose-specific SAT1 knockout mice develop late-onset obesity.
Loss of FTO in adipose tissue decreases Angptl4 translation and
Adipose-specific SAT1 knockout mice develop late-onset obesity.
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